Andrew M. Kazama
Emory University, Department of Psychology
Yerkes National Primate Research Center, Division of Developmental Cognitive Neuroscience
Office: 366 Psychology Department
Additional Contact Information
Department of Psychology
36 Eagle Row
Atlanta, GA 30322
Dr. Andy Kazama was born in Juneau, AK. He grew up on his family’s fishing lodges where he developed a love of nature and biology. His undergraduate work was completed at Davidson College in NC, where he majored in Biology/Neuroscience. Dr. Kazama then moved to Houston Texas, where he discovered his intolerance for high temperatures, but also his love for translational research. He attended UT-Houston, Department of Neurobiology and Anatomy (Ph.D. Track) under the guidance of Dr. Jocelyne Bachevalier, and after one year, the Bachevalier lab moved to Emory University, where he finished his degree in Psychology (Animal Behavior and Neuroscience). Dr. Kazama has dual appointments in Emory’s Psychology Department, as well as the Yerkes National Primate Research Center in the division of Developmental Cognitive Neuroscience, where he conducts translational research focused on understanding the neurobiological basis of PTSD.
PSY 110: Introduction to Psychology I – Psychobiology & Cognition
PSY 385: Neurobiology of Post-Traumatic Stress Disorder
PSY 494: Psychology in the British Isles (Scotland, summer)
PSY 322/NBB 370: Neurobiology of Learning & Memory
My primary research interest lies in better understanding the neurobiological basis of safety signal processing, which is implicated in several anxiety-related neuropsychiatric disorders. The scope of this research includes: investigations of the various brain structures thought to be involved in these processes, the developmental progression of these abilities, and more recently collaborations with Drs. Mark Wilson, and Mar Sanchez, and others, investigating the role of diet and socioemotional factors contributing to emotion regulation.
In lay terms, I am interested in better understanding the areas of the brain that provide a feeling of being safe; something that people that are suffering from anxiety disorders like PTSD find extraordinarily difficult. Understanding how this system works, as well as what factors may be impacting it will open up improved methods of treatment as well as preventative measures for these debilitating neuropsychiatric disorders.
Kazama, A.M., Davis, M., Bachevalier, J. (2014) Neonatal lesions of orbital frontal areas 11/13 in monkeys alters goal-directed behavior but spares fear conditioning and safety signal learning. Frontiers in Neuroscience, 8:37. Doi: 10.3389/fnins.2014.00037. PMID: 24624054
Kazama, A.M., Bachevalier, J. (2013) Effects of selective neonatal amygdala damage on concurrent discrimination learning and reinforcer devaluation in monkeys. Journal of Psychology and Psychotherapy, S7: 005. Doi:10.417/2161-0487.S7-005. PMID: 24567865
Blue, S.M., Kazama, A.M., Bachevalier, J. (2013) Development of memory for spatial locations and object/place associations in infant rhesus macaques with and without neonatal hippocampal lesions. Journal of the International Neuropsychological Society, 19(10): 1053-64. Doi: 10.1017/S1355617713000799. PMID: 23880255
Kazama, A.M., Schauder, K., McKinnon, M., Davis, M., Bachevalier, J. (2013) A novel AX+/BX- paradigm to assess fear learning and safety-signal processing with repeated-measure designs. Journal of Neuroscience Methods; (2): 177-83. PMID: 23376500
Christianson, J.P., Anushka, B.P., Fernando, A.B.P., Kazama, A.M., Jovanovic, T., Ostroff, L.E., Sangha, S. (2012) Inhibition of fear by learned safety signals: minisymposium review. Journal of Neuroscience; 32(41):14118-24. PMID: 23055481
Kazama, A.M., Heuer, E., Davis, M., Bachevalier, J. (2012) Effects of neonatal amygdala lesions on fear learning, conditioned inhibition, and extinction in adult macaques. Behavioral Neuroscience; 123(3):392-403. PMID: 22642884
Kazama, A.M., Bachevalier, J. (2012). Preserved stimulus-reward and reversal learning after selective neonatal orbital frontal areas 11/13 or amygdala lesions in monkeys. Developmental Cognitive Neuroscience; 2(3): 363-80. PMID: 22494813
Jovanovic, T., Kazama, A.M., Bachevalier, J., Davis, M. (2012) Impaired safety signal learning may be a biomarker of PTSD. Neuropharmacology; 62(2):695-704. PMID: 21377482
Bachevalier, J., Machado, C.J., Kazama, A. (2011) Behavioral outcomes of late-onset orbital frontal cortex (areas 11/13) lesions in rhesus monkeys. Annual New York Academy of Science; 1239:71-86. PMID: 22145877
Alvarado, M., Kazama, A., Zeamer, A., Bachevalier, J. (2010). The effects of hippocampal damage on tests of oddity in rhesus macaques. Hippocampus; 21(10):1137-46. PMID: 20882541
Machado, C.M., Kazama, A.M., Bachevalier, J. (2009) Impact of amygdala, orbital frontal or hippocampal lesions on threat avoidance and emotional reactivity in nonhuman primates. Emotion; 9(2): 147-63. PMID: 19348528
Kazama, A., Bachevalier, J. (2008). Selective aspiration or neurotoxic lesions of orbital frontal areas 11 and 13 spared monkeys’ performance on the object discrimination reversal task. Journal of Neuroscience; 29(8): 2794-804. PMID: 19261875
Kazama, A. (2004). Book Review: The Compleat Academic: A Career Guide. Journal of Undergraduate Neuroscience Education; 2(2): R8-R9.